This is how my father died today. He has been dealing with kidney failure and simply refused to do his peritoneal dialysis treatment. He did not want to live anymore due to all of the pain it caused him. He was very delirious, weak, tired and cold throughout the ordeal. He went into cardiac arrest in the ambulance and died upon arrival to the hospital. High potassium is deadly and anyone dealing with hyperkalemia should take it very seriously.

My issue is: Why aren't we warned earlier than Stage 3 that our potassium is inching up each time we have lab work? Many patients in my support group are VERY angry that we aren't notified as soon as any increase in potassium, phosphorous, A1c, etc., shows an ongoing increase even if a little bit. All they ever warn us about is B/P and want to prescribe statins. No mention of anything else.

thanks for posting these! very helpful in anesthesia applications!

Clear, interesting and easy to follow....Thank You!

We had this topic last Spring and this video was a great refresher. Thank you!

@Medcram

9 жыл бұрын

wvllanner4 Good to hear- thank you

Thank you so much !!! I would love if you completed the series with Hypokalemia !! Thanks Again !!!

Cushing’s was an error, he meant Addison’s disease. Thank you for your video.

@Medcram

5 жыл бұрын

+megaroozi correct Addisons is the disease when the adrenal gland is less secretory not Cushings

Thank you Dr. Seheult.

It really is explained clearly! Thanks

Great Lecture !!! Thank you

Thank you medcram

Sooooo helpful!! Thank you 😊

Thank you for the video, very informative

Could you please unlock the treatment video? It’s currently listed as private. Thank you

Can you please complete this series by adding Hypokalemia? Thank You!

Great video! I was a little confused at the end, does hyperkalemia result in the heart cell being more easily depolarized or more difficult to depolarize?

I discovered another helpful channel

Thank you so much 😭

Thanks a lot a great explanation

excellent videos. you do indeed explain things clearly. This is great review for a practicing RN.

@Medcram

8 жыл бұрын

+Navid Aliaghai Good to hear- thanks so much for the comment

exteremly good and simple to understand thanks

excellent video

What happened to the treatment video of Hyper K?? There was a video explaining treatment interactions at a physiological level.

Can you explain how the narrow and peak T wave happen?

Thanks for Good Explanation :) So with Hyperkalemia will become more Depolarized at RMP?

I see how this works. A loss leader video to buy video 2 on the websight.

Thanks Dr. Seheult. Explained clearly. Could you make some video about magnesium, another essential element for our body.

Thanks that's what i was looking for 😊😊

@Medcram

4 жыл бұрын

:)

Excellent video! But still I have a question. At 11:03, did you mention the membrane excitability will DECREASE? If hyperkalemia elevated the baseline potential, shouldn't the excitability be INCREASED? Thankyou.

@kumaradarsh3983

5 жыл бұрын

Increased potassium levels cause a sustained increase in membrane potential and it becomes less negative. One would expect that this would make depolarization easier and the cell hyperexcitable BUT actually sustained increase in membrane potential closes the deactivation gate of sodium channels and this prevents sodium influx and action potential cannot be generated. This is called ACCOMMODATION.

Can you do a lecture on hypokalemia?

Thank you T_T

I am a student and your videos help me. All your videos are so clear and interesting !! Thank you so much !!

@Medcram

9 жыл бұрын

+Abdelhamid Benzerroug Thank you for the feedback

Great lecture but I have a pressing question, can you please tell me the name of the song that is at the end. :)

How does a less negative resting potential cause a spike T wave though? there should be less electrical potential to drive a large influx of K.

I am a student here in the Philippines.. thanks for this video.. it helps me a lot for my discussion..

@Medcram

8 жыл бұрын

Thanks for watching in the Philippines. Best wishes with your studies and thanks for the comment

Thanks, this is a tricky subject :)

@Medcram

9 жыл бұрын

Anete Libeka Glad the video helped

@charleslangdonbostonphilad765

8 жыл бұрын

+MEDCRAMvideos thanks man could u explain why hyponatremia,hypoglycemia and metabolic acidosis occur in adrenal failure

@amineaboutalib

4 жыл бұрын

all hydroelectrolytes disorders basically

See the whole series at www.medcram.com along with other top quality videos including reviews in pulmonary, cardiology, infectious disease, and hematology!

thank you , I suggest to have a lecture on action potential and anti arrythmics , thanks again .

@Medcram

8 жыл бұрын

+‫رصاصة الرحمة‬‎ Thank you for the topic suggestion

What about a burning left arm ? Going to the doctors soon but I’ve had this for about 3 weeks while on keto . I think it’s from high potassium

Great explanation 👌🏽

@Medcram

3 жыл бұрын

Glad it was helpful!

At 5:44, you write "adrenal failure (Cushings Disease)". I think it should be Addisons disease? To my knowledge, Cushings is the opposite to adrenal failure; the cortices are hyperactive, leading to high levels of cortisol and aldosterone, which in turn leads to hypOkalemia, not hyper.

Other than Angiotensin II and adrenocorticotropic hormone, what else triggers the adrenal gland to secrete mineralocorticoids? I didn't even think adrenocorticotropic hormone did this, I thought it only stimulated the release of cortisol. Please explain thank you

Can you explain clearly on the point why hyperkalimia can make depolarization late than normal?

Amazing videos!! Can you touch on some of the other electrolytes. Na+, calcium, magnesium etc. That would be very helpful.

At 12:20 I think you meant to say Addison's disease. Thanks for your videos.

@Medcram

9 жыл бұрын

Ron Norris Thanks for catching this- you are correct. I've made a note on the video that I should have wrote Addison's.

You also get an absent p wave right?

Thanks

I would add rapid infusion or overdosing of IV Potassium as a cause of hyperkalemia. Thank you for the video!

is there any of hypokalemia?

But why do we have a high and fast repolarisation if the extracellular K+ -Concentration is already so high?? I thought also repolarisation will happen very slowly?

@Ani.DR.07

7 жыл бұрын

alsh123am yes same question here.#Medcram plz solve it @medcrAm

@dp7957

3 жыл бұрын

One of the potassium currents (Ikr), located on the myocyte cell membrane, is mostly responsible for the potassium efflux seen during phases 2 and 3 of the cardiac action potential.10 For reasons that are not well understood, these Ikr currents are sensitive to extracellular potassium levels, and as the potassium levels increase in the extracellular space, potassium conductance through these currents is increased so that more potassium leaves the myocyte in any given time period.10 This leads to an increase in the slope of phases 2 and 3 of the action potential in patients with hyperkalemia and therefore, to a shortening of the repolarization time. This is thought to be the mechanism responsible for some of the early electrocardiographic manifestations of hyperkalemia, such as ST-T segment depression, peaked T waves, and Q-T interval shortening from: www.ncbi.nlm.nih.gov/pmc/articles/PMC1413606/

Do you have a video on hypokalemia too?

@Medcram

8 жыл бұрын

+miranda ti Not yet...

Hyperkalemia simplified!!

Why there is a hyperpolarization due to inactivated sodium channels, thanks

@inyeneakpan3612

5 жыл бұрын

@miranda ti please did you get a reply, I'm stucked here too

Is cushing disease causes hypokalemia or hyperkalemia ?

it is said that hyperkalaemia is often associated with metabolic acidosis.... yet the only cause in which acidosis and hyperkalaemia occur is addison's disease. other causes of metabolic acidosis ( DKA, acetazolamide, diarrhea, and renal tubular acidosis are associated with hypokalaemia. would you like to explain that please??

@Medcram

6 жыл бұрын

+Nada Dwiddar this is because acidosis itself causes hyperkalemia. Protons in the serum go into the cell in exchange for potassium which comes out of the cell causing hyperkalemia.

Very informative! Thank you so much! 😃😃 27/2/2019

Adrenal failure is Addison disease not Cushing sir

Is 3.47 level is dangerous what to do on this level.

@sassydesigngirl

4 жыл бұрын

No thats normal

can u expalin the relationship between k level and acid base balance..... very confusing

@Medcram

7 жыл бұрын

+Nada Dwiddar yes as the blood pH goes down this causes protons in the blood to go into the cells. Potassium then comes out of the cells to balance it and this causes hyperkalemia. The opposite occurs when the blood pH goes up.

@nadasdwiddar

7 жыл бұрын

MedCram - Medical Lectures Explained CLEARLY thank you very much..... 😊😊

why a grain of salt if hemolysis occurs?

Dude, you sound EXACTLY like Sal Khan...unless you narrate the MCAT videos on Khan Academy?

I think der r some errors here.. Firstly..tendency to depolarise increases initially bcz K conc outside the cell becomes high nd hence its difficult fr K to leave the cell nd hence the polarity inside bcmz more postve nd towrds the threshold potential..bt then free flow of Na should happen and it wont happen bcz f increased postve charge inside the cell..this will actually delay the depolarisation.Now during the repolarisation, initially der s a plateau phase where K efflux and Ca influx happens simultaneously..but since K does not leave as easily as expected bcz f reduced gradient,there will be prolongation in the repolarisation which is evident from the delayed or wide QRS..Eventually leading to absent atrial depolarisation and sine wave pattern nd ventricle fires at its own rate and produce ventricular fibrilations nd cardiac arrest @ d end evidenced by asystole in ECG

But why Does acidosis cause hyperkalemia ? This remains my question :p Maybe you can clarify for us sir ?

@hattietoodle7496

7 жыл бұрын

pulmonary embolism

Watch at 1.5 speed... you're welcome

Adrenal failure is known as Addisons Diseases, not Cushing’s syndrome.

a very informative topics

my dad had hyperkalemia and doctors placed a pacemaker in his neck

I’m 24 but suffering hyperkalemia rn 🥺

🥰

Dialysis patients should never use salt substitute.

Tall tented t wave.. hyperkalemia..

Not so helpful.... 6/10...only

WAY too scientific for most viewers to understand... this sounds like a doctoral dissertation!!

Can you do a lecture on hypokalemia?

@Medcram

8 жыл бұрын

+Hashim Ismail Thanks for the suggestion

@evasmiljanic3529

8 жыл бұрын

seconded

@carvchlo

8 жыл бұрын

We are waiting :) Thanks so much!

@TheGreatSniper

8 жыл бұрын

Preferably before my nephrology exam on the 31st of August, please :P

@nellyhoffman6194

7 жыл бұрын

PLease did you do it ?