Wow....finding this after 5yrs and it was exactly what I needed! Thank you!

I'm always so glad when I find one of your videos with a concept I need help with!! They're incredible. Great job!

I can't say how much I appreciate your video. Your explanation is so clear. Thank you!

Simple easy and crystal clear and no time wasting at all.I took GoodmanGilman pharmacology text book to clear the doubt,but then out of mere curiosity I searched on KZread which ultimately ended up in your video and the doubt too ended in a very short time.thanks a million times for saving our time and suffering

you always give me hope in life ... many thanks

No other video or book has explained this topic so well! Thank you so much for putting this out there!!!!

Dr. Meet is the best. :)

Extremely helpful, thank you for taking the time

Very well explained. Best thing is u try to make our concepts clear so Thankyou very much. U should keep on making more videos. Prayers for u

im so glad i found this, thank you so much!

Wow so helpful and clear! Thank you 👌🏼😊

Hi, I avoided watching this video at the beginning & watched almost ALL videos about this topic but your explanation was PERFECT (I wish I watched it from the beginning). I fully understood it although I haven’t started cardiology (just going through pharma)

you are so helpful thank you!!!!!

Thanx..great help..

MASSIVE THANKS!!

Thanks a lot!!

Thank you, very helpful!

Soooo helpful,thanks alot🙏🏻🙏🏻🙏🏻

Thank you so much !!

Bless you

Great work 😊

Thank you!

Really helpful 👍 ty so much

was a very detailed explanation of the graphs :) Thanks a bunch Dr.Meet:)

@Meetpatel-pu6me

7 жыл бұрын

thank you dr. nishi :)

Less time consuming and perfect explanation

Good job Dr.Meet, very helpful, keep it up

@Meetpatel-pu6me

7 жыл бұрын

thank you :)

Thanks

I wish i could like this video twice

🎉🎉🎉

❤️

Love your Indian accent ♥

Thankyou!!

this was -rea8lly helpful thank you

Well explained but i have a question regarding norepinephrine is that if it is causing reflex bradycardia wont systolic pressure decrease ?

I have a doubt sir.afterload depends on tpr too right.so alpha1 stimulation increases tpr increasing after load...hence decreasing systolic pressure.so like cardiac output remains normal in norepinephrine...systolic and do the sane too right..

Sir after alpha blocker if we give phenylephrine then as phenylephrine has selectivity for only for alpha 1 then after its blockage it should DBP should decrease..but u show that it is increasing why ????? Plz plz ans ASAP

cardiovescular drugs antihypertensive heart failure ke send kro..please

Hello. Thank you for your videos. Could you please do a video on open and closed angle glaucoma? Could you explain why a Muscarinic antagonist, such as atropine, can cause acute angle-closure glaucoma? It is on page 245 of 2016 FA? Many thanks.

@Meetpatel-pu6me

7 жыл бұрын

Thank you :)) . I want to make video on this but time and situation currently is not permitting me. I will try to make soon.

@karthikeyareddyramasahayam1039

5 жыл бұрын

the way i understood may hepl u ,sry if im not correct:M antagonist cause mydriasis thru radial muscle contraction and cycloplegia(DUE TO BLOCKING THE ACTIVITY OF M3 RECEPTORS).AS these actions are on iris muscle,cycloplegia amd mydriasis have effect on angle decrease (ANGLE DECREASES MAY LEAD TO GLAUCOMA)between iris and cornea that results in reduced drinage of aqueous fluid(rate of prduction is constant) result im incresed IOP

Sir Goodman writes with norepinephrine CO either remains same or decrease not same or increases

@Meetpatel-pu6me

4 жыл бұрын

Because of alpha-1 mediated venoconstriction the preload will increase and there will be beta-1 receptors stimulation as well. Both these factors leads to increase in CO. Although, as the venoconstriction increases MAP, there might be reflex bradycardia later on and that's what they might be mentioning. But from all the texts that I've read it's mostly 'remains same or might increase'. I hope this helps!

Great Job and Teaching style. Would you consider uploading a scanned copy and sharing your high yield notes? Looking forward to more videos, thanks!

@Meetpatel-pu6me

7 жыл бұрын

Ali Anjum thank you....i dont have pre made notes

God bless u.. why do MAP decrease in isoprotrenol?!

@Meetpatel-pu6me

7 жыл бұрын

Sara Rp thank you. MAP = 1/3 SBP + 2/3 DBP. so even if increase in SBP and decrease in DBP is same still MAP will decrease.

@es3259

6 жыл бұрын

Map decreases bcz of B2 effect (vasodialtion )

Hello! I have a question. For isoproterenol, why is there a net decrease in the MBP? if B1 and B2 are equal, i wonder why the BP isn't cancelled out.

@pallavisharma6000

3 жыл бұрын

In periphery b2 are more.. now due to selectivity of b2 of isopretenol DBP decrease very much..due to which MBP also decrease...

@pallavisharma6000

3 жыл бұрын

Now HR increase because sir has told that if any drug has selectivity for beta receptors it will automatically increase HR no matter what was MBP

Why NE can increase SBP than Epinephrine even though they both have alpha1 and beta 1 effect?

@rinc2006

4 жыл бұрын

Because norepinephrine has no beta2 action which is dilation of blood vessel in the skeletal muscle

Why does MAP increase with epinephrine?

@umerbinshabir6561

4 жыл бұрын

Because systolic pressure increases

Don't know if you'll see this, but doesnt epinephrine and isoproterenol cause increases in heart rate because they are beta 1 and beta 2 agonists? You keep saying its because of beta 1 effects, but norepinephrine also works on beta 1...

@SuperHaind

5 жыл бұрын

Epinephrine works more on B1 >B2>alpa 1 , so its effect will be more on heart rate and vasodiation than vasocostruction. On other hand, norepinephrine works more on alpha 1 than B1 so will has effect on vasoconstriction increase Dbp.

FA2022 pg no 244

00:40 spider ! :)

@Meetpatel-pu6me

3 жыл бұрын

Baby cockroach ! ;)

Reason for changes of map ?thank u 😊

@Meetpatel-pu6me

7 жыл бұрын

MAP = 1/3 SBP + 2/3 DBP. so whenever SBP or DBP changes, MAP changes.