As someone with High Lp(a), this was the best source of info on the topic I have seen. Thank you for a great presentation

Thank you for the webinar. I shared it in my lpa/ heart attack Facebook groups

Lp(a) is a risk factor, not an absolute sentence to cardiovascular disease. My doctor says "80% of CVD is environmental." So, as the video says, don't smoke, avoid obesity and diabetes, don't eat junk food, exercise, etc. Plenty of people with high Lp(a) values (which supposedly is inherited in a co-dominant fashion) never develop CVD nor have their parents and other close relatives.

@sheddkkhan6758

2 ай бұрын

Are you busy

@JaySeitzPhD

13 күн бұрын

What's the evidence for this statement: "Plenty of people with high Lp(a) values (which supposedly is inherited in a co-dominant fashion) never develop CVD nor have their parents and other close relatives."

@danielevans5864

9 күн бұрын

Plenty? Quantify that description, please.

Yes, statins increase lip (a) significantly for people with genetic abnormalities, for example - family hipercholesterinemy

I was to be in a trial to reduce LPa (mine was in the 245 range). The followup prior to be in the trial my LPa was at 150 which was to low to be in trial. The only thing that changed was I was put on Repatha maybe 3 weeks before the followup. 150 is still high but it's been in that 240..250 range for about 10 years.

Thanks for your presentation. As far as LP(a) levels it seem that the measured levels can differ when considering either mg/dl or nmol/L, they appear not to be interchangeable although 2.5 is being used as a converting number. Any comment on that? Thanks again

I have it -- it is over 1500 so I am doomed sadly - live life

There are some old studies showing that androgens (testosterone, anabolic steroid) lower lp(a) by 60-80%. People who take testosterone replacement or anabolic steroids could be benefiting from it. Has anyone studied this?

@whobdis77

19 күн бұрын

I can tell you it didn't lower mine..I've been on TRT for a year and LPa is about the same.

Dr. Khandelwal, are you familiar with the Lp(a) work done by interventional cardiologist, Dr. Nadir Ali, from Webster, Texas? Dr. Nadir Ali not only opines that Lp(a) has NOTHING/ZERO to do with CAUSING atherosclerotic diesase (CAD, CVD), but he describes Lp(a) as a very important molecule in the repair process of damaged tissue. Dr. Ali describes some elderly patients, with lifelong high Lp(a) levels, who he has performed coronary angiograms on, and these patients have absolutely NO plaque whatsoever.

@quality6823

3 ай бұрын

My father,his 3 sisters and a brother all passed away av a kind of fast and powerful heart attack at their late 50s. All their children have extremely elevated LPa. Mine alon is 469 nmol/l. So, your Doctor probably has done his research on wrong people.

@petraharms194

3 ай бұрын

Das wäre großartig. Ich habe im April Herz Ct mit Kontrastmittel. Ich bete,dass ich keinen Plaque habe 🙏🙏Danke für das Video

@alexandrastevens8892

3 ай бұрын

My LPa is 114, and I was told by BHF that in there opinion up to 300 is not considered extremely high and enough to impact on coronary health. But over 300 should be managed by lowering LDL to help stop the LPa being able to stop it travelling round the body. If its so important why isn't this test being carried out on everyone who has the cholesterol test.

After rosuvastatin I have lip (a) 517. I will try PCSK9 inhib.

@sharonillenye8036

13 күн бұрын

I just found out mine is 486nmol/L after 4 years on a statin and then went off of it. I wish I had measured it before the statin

After six weeks of 2000 mg per day Niacin treatment my Lp a dropped from 147 nmol/l to 45. I begun another six week 1000 mg Niacin treatment.

@inaratolocko5723

22 күн бұрын

How you feel yourself using 2000 niacin?

@edwrobel

22 күн бұрын

Little sweat at night, somethimes

@edwrobel

20 күн бұрын

@@inaratolocko5723 very well. I should have new blood test results this week. I highly recommend Niacin, inexpensive and very effective

@edwrobel

20 күн бұрын

And again after six weeks of 2000mg Niacin per day result: 48 nmol/L

@edwrobel

19 күн бұрын

My new result is 48nmoll/l. Ecellent!

Can it cause pulmonary embolism in lungs?

What role does ethnicity play in disease risk? Are elevated LP(a) levels as relevant for African Americans, as the Caucasian population?

Best treatment for atherosclerosis: Vitamin C Why do only coronary arteries clog with cholesterol and not veins or small capillaries? Scurvy = No vitamin C in your diet. Symptom: Your blood vessels break and you bleed to death. Think of the sailor of the past. Scurvy of the heart = Just enough vitamin C from food. Symptom: Arteries around the heart are not strong enough to resist high blood pressure. Damage is repaired with cholesterol LP(a) to prevent worse. After years of repair, your arteries become clogged. Enough vitamin C supplement of at least 3000 mg. per day gives strong and flexible arteries. Cholesterol is not needed as a repair agent. Cholesterol in your coronary arteries is broken down and burned in your liver. Source KZread: Cardiovascular disease and vitamin C (Dr. Rath Foundation) or Breakthrough Towards The Natural Control Of Cardiovascular Disease - Dr. Rath's 2018 Cyprus Lecture