Amazing information! Thanks Tamron & Dr. Sanjay!

If only all children could be around for their senior parents when they really need you.Sad when they are not shameful really.

What is the name of the book TAMRON??????

@SoTiredoftheToxicBS

2 ай бұрын

Keep Sharp.. is the name of the book.

I am ordering that book today! My father passed away 3 years ago and he had developed Alzheimers. I am worried about it as I am aging. 😞

@salyol7

2 жыл бұрын

Live life do not think of how your future may be.That brings anxiety.

Am I the only one who can’t believe she is 50 .. wow looks 30

My stepfather died with Alzheimer's, as did two of his brothers and his sister, so I find it hard to agree with the title of this clip. However, I did appreciate the informationon how to protect myself from getting the non-hereditary version!

My paternal great grandmother, my grandmother, all 3 of my dad's siblings and finally, my dad all passed away from Alzheimer's. I believe it is hereditary.

Very misleading. Neurogenesis in adult brain is limited to two small areas and there is no evidence that the new cells are functional. And... genetics play a very important role. Individuals with two APOE-4 alleles have a high probability of developing Alzheimers regardless of lifestyle; though that is not to say optimized lifestyle won't decrease that probability somewhat.

@MoreThanOrder

2 ай бұрын

“High probability” without statistical data is misleading. INFORMATION ABOUT THE APOE GENOTYPE AND ALZHEIMER'S DISEASE Alzheimer's disease (AD) is the most common form of dementia in the elderly and currently affects more than 5 million Americans. It is a progressive neurodegenerative disorder with brain findings of plaques and neurofibrillary tangles containing beta-amyloid and tau protein respectively. 🔻The predominant form of AD is late onset (age > 60-65), which can be familial (15-20%) or sporadic. 🔻The APOE4 variant increases the risk for late onset AD and may contribute to the pathology of the disease. This risk is increased by approximately 2 to 3-fold for individuals with one copy of the APOE4 variant and by approximately 10 to15-fold for individuals with two copies of this variant (E4/E4 genotype). The APOE2 variant has some protective effect against development of late onset AD. 🔻The lifetime risk for late onset AD is approximately 10-12% in the general population, though it is higher in women than men and doubles when there is a first degree relative with this disorder. The lifetime risk is approximately 9% for individuals negative for APOE4, and for individuals with E4/E4 may be as high as 25% for males and 45% for females. Among patients with late onset AD, the presence of APOE4 may lead to earlier development of symptoms. However, APOE4 is neither necessary nor sufficient for the development of AD. Approximately 30-50% of patients with late onset AD do not have an APOE4. 🔻APOE4 is common, with 25% of the general population having one copy and 1% having two copies of this variant. Among patients with late onset AD, 50-70% are positive for APOE4. The development of late onset AD is influenced by many factors other than APOE4 including age, gender, family history, level of education and history of head trauma. Midlife cardiovascular risk factors in individuals with APOE4 also increase risk for cognitive decline. A number of genetic influences in addition to APOE4 h ave also been reported and are under investigation.

@lisa tanner yes but many of the hollywood AFRICAN AMERICAN celebrities dont look black they look light skin & mixed race.ebony mag says they use this lotion fer.la which makes them look far more light skin & mixed race and not black anymore. Im not surprised everybody seems to change their appearance these days.