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  @stephenmcconnell1935Ай бұрын

  Sharon, essentially YES - 16% of ApoB + Lp(a),.. is one of the highest percentiles,...

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  @rajthapa1997Ай бұрын

  Very informative. Thank you

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  @luckssjАй бұрын

  I used Magnesium Glycinate and Liposomal Vitamin C witch prevent LP(a)

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  @a32003Ай бұрын

  Sir thanks for your priceless informative lecture you presented. May I ask how we made a balloon to be probably cutting for distal pressure measurement?

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  @hikmatyar5416Ай бұрын

  Excellent video. Covered all bases and and a great primer for new fellows starting cath. As someone who was trained in the radial first doctrine I must say your video is quite comprehensive! Great job

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  @sharonillenye8036Ай бұрын

  if my LPa is 486nmol/L (which it is) and my apo B was 160mg/dL = 2909nmol/L. Is this correct? and does that mean that one out of 6 of my LDL is the LPa type?

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  @sheddkkhan67585 күн бұрын

  Hi

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  @theluckyman742 ай бұрын

  It is all in circulation or also in the heart if a person receives a new heart transplant could it also be there. I would imagine only in the circulation

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  @leandrobecker1233 ай бұрын

  Excelent class! Congratulations

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  @jimmcnair85033 ай бұрын

  Excellent thank you

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  @draksingh80343 ай бұрын

  Thank you for your presentation Gabriele. It was very nice to see your confidence in performing the whole PCI with balloon inflated.

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  @obscuraterror18913 ай бұрын

  Great!!!

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  @vladimirandrianov59303 ай бұрын

  Thanks a lot for sharing this content! The infromation presented here is inavuable for us people with hyper-lp(a)-aemia, for making informed decisions about our health and medical interventions. Especialy when official recommedations are obviously lagging the science.

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  @tracyeeadesmickle60903 ай бұрын

  I jave a 25mm Gorman Occulator PFO closure.

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  @edwrobel4 ай бұрын

  I lowered my Lp a from 147 to 45 with niacin 2000mg per day for six week

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  @torpol4 ай бұрын

  Did you take it all in one dose? or spread out over day?

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  @edwrobel4 ай бұрын

  1000mg early morning and 1000 at night. Now 500mg in the morning and 500mg at night.

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  @torpol4 ай бұрын

  Thank you!!

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  @edwrobel4 ай бұрын

  You are welcome.

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  @drironmom68153 ай бұрын

  Did you experience any side effects from niacin? And did you take plain niacin or extended release or “non flushing” niacin?

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  @jacksonkansiime17084 ай бұрын

  Why did you give him gloves? It was his own heart

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  @drironmom68153 ай бұрын

  lol I thought the same thing. Maybe it was already soaked in formaldehyde

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  @victorblock34214 ай бұрын

  Such a great man, Dr. Moses. He worked on my dad when every other doctor said stuff like "oh we'll adjust his meds and he will be alright". He saved my dad.

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  @MuhammadUsman-zo6oz4 ай бұрын

  Excellent ❤

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  @BlackMamba-fb3wg5 ай бұрын

  Thanks for sharing.

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  @user-ef4wl2rr1k5 ай бұрын

  Even non-physicians can follow and understand this clear, informative and interesting lecture. I am honored to have Dr. Iacobellis as my physician.

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  @Anonymous786695 ай бұрын

  This guy is from AIIMS Delhi.

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  @varunsinha19685 ай бұрын

  Excellent clear presentation!

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  @munirabdullatif20225 ай бұрын

  Great webinar, thank you.

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  @Joeythegoats6 ай бұрын

  25:00

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  @svijaykumarreddy12426 ай бұрын

  Excellent talk

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  @michaelmindrum50387 ай бұрын

  Nice lecture and overview Dr Albert

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  @ahtishamshakoormd7 ай бұрын

  Excellent talk

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  @leandrobecker1237 ай бұрын

  Very good class dr. Miguel. Thanks also Leandro. Here in Brazil cac score and coronary CT are progressive more used !

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  @leandrobecker1237 ай бұрын

  Very good class! I do agree that plaque quantification and classificatoon by CT must fast be in any report for us as clinical cardiologists.

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  @IlIKRATOSIlI7 ай бұрын

  So if Lp (a) is raised by other factors not including FH and APob100 then is it still a cardiac risk or simply an immune response?

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  @vladimirandrianov59303 ай бұрын

  if it's not fh then it must be transient and therefore not pose a cardiac risk

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  @lorrainevale97489 ай бұрын

  I'm in the UK and will be having this procedure 30th Nov 23. I have had issues with the heart since 2008 including heart bypasses. At the moment I can't do anything under pressure I have a angina attack. I'm scared about having this it's most probably the fact it's done through the main vein in the neck. I had stents through the groin. Can someone put my mind to rest a little please.

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  @renus60159 ай бұрын

  Informative....

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  @user-od7wc9ki3w9 ай бұрын

  Great feedback discussion. I also support the view that hsCRP >2 should NOT be used to select patients who would benefit from colchicine for all the reasons you mentioned. Further, it would exclude its use in almost 2/3rds or patients with chronic coronary disease who have the same atherosclerotic process as those with a lower hsCRP at baseline and have a life time risk from their disease. While hsCRP is sensitive to risk in a cohort, it lacks specificity when assessing individual risk. To not prescribe colchicine in patients with proven coronary disease would be akin to not prescribing a statin in patients with a low LDL. Lowering LDLc and dampening inflammation are 2 sides of the same coin and both must targeted. Besides, 0.5mg of colchicine is safe, with a low risk of drug-drug interactions in patients without renal disease, and inexpensive (outside the US).

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  @radiant20123 ай бұрын

  My (new) cardiologist will not prescribe Colchicine for me. The dr I had practiced preventative medicine and he was “let go”. I am that patient you describe & I am confounded by this Drs actions and words, I am in the process of finding a new doctor. Ps.. I don’t have renal disease nor kidney issues.

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  @paddainstitute10 ай бұрын

  I believe the Slide on Topiramate is wrong at the 32:10, Topiramate enhances GABA (A) receptor activity, which inhibits the AMPA and kainate subtypes of glutamate receptors. The Slide states the opposite. Topiramate is an anti seizure drug, activating the Glutatmate pathway ould trigger seizures.

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  @Itsme-jv4cd10 ай бұрын

  This is the most thorough and interesting lecture I've heard on cardiac amyloidosis. It should be required by all cardiologists.

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  @grantingapex816910 ай бұрын

  Thank you. Just enrolled in local study. Great presentation.

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  @svijaykumarreddy124211 ай бұрын

  Really amazing

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  @Nikesnipe11 ай бұрын

  Great Lecture! Greetings From Germany!

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  @MikaelVitally-rf5pn Жыл бұрын

  High blood pressure is the symptom for a medical problem. Artificially lowering blood pressure with drugs does not treat the "root cause" .While medication can control blood pressure, it cannot cure the condition.

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  @amberdawn7426 Жыл бұрын

  😮 my aunt was Diagnosed with it and died at 55 from Liver Cancer. My Dad shows symptoms as well and has major issues. I have red Spots and Kidney issues. I just researched up testing centers.

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  @farrokhfarr2694 Жыл бұрын

  What do you think about statins?

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  @kainataslam3999 Жыл бұрын

  Great lecture ❤

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  @nolaemerson5074 Жыл бұрын

  Promo`SM ☹️

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  @PlantBasedPrimary Жыл бұрын

  Doctors: Please listen! Prevention may not be as lucrative as the allopathic pharmaceutical/ procedure model, but it is incumbent on you to inform your patients of the benefits of a whole food plant based diet.

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  @user-ij9jk3so2m Жыл бұрын

  Great information, Thank you! Especially for the great information on LP(a). Many of my family members have very elevated lp(a) (200+nmol) and we are anxiously waiting for Pelacarsen results and approval.

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  @SteppesoftheLevant Жыл бұрын

  More cardiac arrests in the last 3 years. Wonder why

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  @slimshady5510 Жыл бұрын

  No one is talking about myochordial bridge?

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  @tarheelace Жыл бұрын

  Excellent.

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  @mcviegas2566 Жыл бұрын

  When Should one consider Amplatzer Occludor removals, & the right fix, seriously?! Why you ask? Well errosion or a compromise of healthy tissue become detached or ruptured?

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  @shashijanjirala1402 Жыл бұрын

  Thanx gazzillion times

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  @MrDarklord91 Жыл бұрын

  If possible can you please go over how the invasive hemodynamics would be different in a patient with HFpEF as compared to cardiac amylodosis