Shock - hemodynamics | Circulatory System and Disease | NCLEX-RN | Khan Academy

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Пікірлер: 13

  • 9 жыл бұрын

    Afterload is not necessarily related to the amount of volume that remains after the squeezing of the heart, it has to do with wall stress that opposes the ejection of blood, or the wall stress at the moment of the ejection.

  • @norah3874

    @norah3874

    7 жыл бұрын

    Jesús Ortiz exactly! I was confused by that bit

  • @LauraMannes
    @LauraMannes5 жыл бұрын

    I think you need to revisit your definitions of preload and afterload!

  • @50BMGdefense
    @50BMGdefense2 жыл бұрын

    Afterload is the amount of pressure the ventricles must overcome to eject their blood, *not* the amount of blood left in the ventricles after contraction.

  • @KJax131
    @KJax1318 жыл бұрын

    At 5:30, you said an increased SVR(vasoconstriction) would increase tissue perfusion? I think you are incorrect as a DECREASED SVR (vasodilation) would increase blood flow thus increase tissue perfusion.

  • @ragu5170

    @ragu5170

    6 жыл бұрын

    Frost Rival you are right .. but you think in other way too.. increased SVR means decreased perfusion .. but during shock not every vessels has increased SVR, only the blood vessels to nonvital organs like skin have increased SVR... But the SVR has no effect or decreased , in blood vessels to vital organs .. so that the perfusion is better in vital organs.. this is how I understand.. hope it may help you

  • @G2H_HellBringer
    @G2H_HellBringer6 жыл бұрын

    I've got a question that isn't related to this topic. What is the hardware and program shown in this video? (the black background, text, etc)

  • @chadb3525
    @chadb35257 жыл бұрын

    CO and SVR are inversely related. That is why CO is high with septic shock, because the low SVR (afterload) creates little impedance for outflow. Afterload and ejection fraction are not the same, which this video claims. The greater the resistance, the harder the heart must work to overcome it. This is a basic principle of how prolonged RAAS activation requires treatment with ACE-Inhibitors. Please redo this video.

  • @ragu5170

    @ragu5170

    6 жыл бұрын

    Chad Backus but he never mentioned that CO and SVR are either directly or indirectly proportional.. he said that perfusion is directly proportional to CO, SVR and O2 content

  • @playmobilandothers34
    @playmobilandothers345 жыл бұрын

    MAP = (CO x SVR) - CVP, not +

  • @punies24
    @punies249 жыл бұрын

    Bb

  • @manjitdhami
    @manjitdhami8 жыл бұрын

    Why 2/3 diastolic and 1/3 systolic, why not 1/2 and 1/2, confused

  • @rashy28

    @rashy28

    6 жыл бұрын

    Manjit Dhami the heart spends twice as long is diastole as in systole

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