Thank you so so much!! especially for the last part. I literally searched the whole internet trying to figure out why patients will be in a flexed position. A life saver!!!!

@mukeshrawat975

4 жыл бұрын

Me too

@fredrikkarlsson1021

2 жыл бұрын

Just a thought, why doesnt the same thing occur with rigidity? Is the agonist and antagonist affected unequally then?

Thank you so much for the explanation. Studied for 4 years in med school and now only I can fully understand the pathophysiology of spasticity in UMN lesions

Took the brakes off my learning curve, thanks, man.

Amazing explanation ❤❤❤

Never made sense to me until now, thanks so much!

You cannot explain this any better - it is perfect - clearly the Gold standard - Thank You sir -

@user-tw7lc1hz9i

2 жыл бұрын

gold standard indeed

Thank you so much! My course lectures didn't explain it as well as you did, and also giving the explanation of the clinical presentation really helped!

Thank you so much Prof ... you made things super easy

Great Explaination. Thank You !!!!

Wow, really good. Saudations from Brazil!

Excellent lecture. Perfectly explained.

freaking brilliant presentation.

Great video, well done.

Help me a lot! Thank you!!!

Heard some "M'kay" here and there, which remind me of Mr. Mackey from South Park :) Jokes apart, wonderful explanation. You really have the gift of teaching!

Well Done! So clear explaination!

Thank you, this was really clear! :)

Great explanation ✌🏼

Awesomely explained!!!!

Thaaaanks..easy and informative.

Thanku 😊clearly explained

Thank you! ☺️

Learn a lot. Thank you!

Thank you so much 🌻

awesome! thank you

Role of gamma motor neurone in development of spasticity is not explained in this video. Other wise the video has good information for students. Thanks for your contribution.

THANK YOU!

thanks a lot Sir!!

Legend

Thank you

Great vid

thank you!

Thank you so much sir this is really helpful

There are some debate on wether spasticity is "just" the change of balance between inhibitory and exitatory stimulus. It doesn't really explain the different types og hypertonia - rigidity or spasticity. Why is it, that rigidity does not show increased reflexactivty

Thank you so much sir.

Thank you for this, but could you explain why streaching the spastic muscle, relaxest both flexors and extensors? im just a student, ive never worked with a spastic pacient, but im very interested!

Thank you sir!!

Would you know why touch helps calm spasticity? Like when touching the fingers with the other hand, the fingers are easier to open

Best explanation

Thanks a lot Sir

Hi Steve, do you know if there is a cure to hypertonia, or at least an effective treatment?

Thanks a lot 👍✅

Thank you , i fully understood your explanations on why this occurs bI'm a stroke survivor who likes answer but can i re wire this within my brain i can move my arm and get it to relax until i move or stand up then bends into the poistin within your picture :

Thanks

Very good video

Can the damage heal either on it's own or with some help?

Sir please make one video on spasticity vs rigidity and also types of rigidity,,,clasp knife ,lead pipe,cog wheel with their mechanisms???

so that's why my left hand can't relax at times (I have cp)

Sir do u mean Lower motor neuron is relatively controlled by inhibitory neurons than excitatory..so removal of inhibitory causes excitation..!?plz explain..

❤️

Great. Why does the spasticity ''loose up'' at the end of motion in contrast to rigidity?

@stevejj14

7 жыл бұрын

Are you referring to 'clasp knife' rigidity, where resistance suddenly 'gives' when enough force is applied? This is caused by Golgi tendon organs, which detect high levels of tension in tendons. They are part of a protective reflex (ever involuntarily dropped a weight that is too heavy for you?). Golgis are connected to inhibitory interneurones in the cord which inhibit motor neurones and cause the give. Hope that helps

CST is excitatory?

@che-lunhu9074

5 жыл бұрын

I think CST neurons stimulate the inhibitory interneuron that connects to the alpha motor neuron (in blue).... Thereby inhibiting/controlling muscle contraction.

@acingmedicine

4 жыл бұрын

@@che-lunhu9074 Corticospinal tract is the descending excitatory tract to the alpha motor neurons to initiate voluntary movement

What part of the brain must be damaged for this to occur?? plz reply

@stevejj14

3 жыл бұрын

Hello. This is caused by damage to upper motor neurones that send their axons down to the spinal cord

How neuron is pronounced is bugging me. I never heard it pronounced quite like that before.

Ventral horn is bigger than dorsal horn correct your drawing

in the UE the flexors are stronger and win out over the extensors. this isn’t very scientific. how would you then explain decerebrate? really need to back up your thoughts with some science references, otherwise this really is just conjecture.

Thank you