Before I started keto my cholesterol was 244. At that time I was eating maybe 2 to 4 eggs a week. The rest were carbs in the morning. After starting keto I started eating 3 to 4 eggs a day. So from four eggs a week total to about 21 to 28 eggs. Also I found a guy in the area that raises chickens and ducks. So I buy his duck eggs which I like even better than chicken eggs. The duck eggs are larger than chicken eggs by about 1 1/2 to 2 times the size. After about a year of eating like this I had my cholesterol checked. It’s still at 244 had not budged. And I’m not concerned at all about it because all my other markers improved dramatically. Cholesterol is the passenger not the driver.

@cinnamongirl3070

4 жыл бұрын

Dave Feldman!!

@gabriel65304

2 жыл бұрын

if you have chicken eggs in the household it is healthy

@DK-pr9ny

Жыл бұрын

Cholesterol number doesn't matter. It's all about LDL particle size.

what a player - very clear, thank you

Excellent! I'm look forward to see more of your lectures Dr. Sikaris

Thank you for your videos. I look forward to the next one because I am a hyper responder but with HDL of 3.5 and TRG of .5, I hope I don't have to worry.

So clearly and precisely explained. An excellent presentation. Even indicating the relevance and importance of the TG/HDL ratio. Thank-you!

Wow! Awesome! Finally all pieces got together! Thank you so much dr Ken!

Thanks for a remarkably understandable overview of so many studies. Our family has FH and prior to my generation a very short lifetime. Everything presented by Prof Sikaris has real meaning. I look forward to the next presentation.

Same. I was interested until he said FH requires statins control. Utter rubbish. I have FH. 19 years ago, my TC, HDH, LDL, Trigs were all so high that I was told to continue taking statins or die within 12 months. But the pains and my avid reading of 'alternative scientific views' encouraged me to stop the medication. I immediately felt better. However, my lipid profile was getting worse and I was worried. Later I discovered LCHF and then KETO. Today, 19 YEARS LATER, I look and feel 15 years younger, my Trigs and CrP are low, I'm no longer even 'at risk'. They think they know, but all they really have are assumptions.

@yoso585

5 жыл бұрын

Antya Markin Perhaps you weren’t FH. Don’t know how they test.

@lamooswa4704

4 жыл бұрын

WHAT DID YOU DO ? WHAT IS LCHF AND HOW HIGH WAS YOUR TRI ?

@BokraElna

4 жыл бұрын

Antya Markin . Yup. Your case is similar to mine. It is now 15yrs since i was discovered accidentally to have a high cholesterol levels. I kept hesitant to take statins and lipids. Probably less than 5 distributed yrs out of 15 out on these drugs. Recently, about 7 months ago I started LCHF diet and my trigrocerids level went very low and HDL improved by about 30% with a rising shoot of LDL. Mu question to you os did you have similar shoot in LDL levels. I haven’t been confirmed as HF anytime. Just guessing from time to time by this or the other doctor. Can you please answer my question about ldl rise after LCHF diet.

@jw-vx8im

3 жыл бұрын

@@BokraElna it's fairly normal

@gabriel65304

2 жыл бұрын

@@BokraElna how much was your ldl cholesterol ? HDL and triglycerides? my cholesterol levels after 2 months of keto diet, august 2021 HDL- 50 LDL - 275 Try. - 85 I have to be worried 😟?? Thank you so much your help 🙏🏻

Excellent!!! I learned a lot from his speech!!

Nice points and very clear explanations

Food for thought. I have been low carb for 3 years and dramatically reduced the usual markers such as inflammation, blood sugar, glucose, weight, etc. Yet, my LDL, small particle number and apo B got to a point where my Dr wanted me on statins. In the last 45 days, I increase my soluble fiber intake to roughly 45 grams daily through psyllium, chia seeds and more fibrous vegetables such as eggplant and okra and my total cholesterol went from 220 to 150 and my LDL went from 155 to 93. This reduction was in the last 45 days alone....

@tanyasydney2235

Жыл бұрын

Excellent. Yes, fiber helps to reduce cholesterol.

@Cenot4ph

Жыл бұрын

as discussed in this video, sdLDL is what should be checked for as a heart disease risk not general LDL numbers

Low Carb Down Under: I've been a subscriber for a while now, great channel mate, well done.

Why don't GPs know this information? All they do is use that risk calculator and tell you're going to have a heart attack in 2 to 5 years. I mentioned ldl subfractions and the GP had no idea what I was talking about. Frustrating.

@joeohalloran9309

4 жыл бұрын

Agree my doctors have no idea. When I talked about small dense LDL she looked confused. Doctor "your total cholesterol is high 6.1 going up go in statins Joe or you'll have a heart attack.

@Erowens98

3 жыл бұрын

Because normal doctors are generalists. They have to know a little about everything. So they can't always get into the gritty details. Since they don't teach this in med schools, the only doctors who know it are the handful who have the time and interest level to do research on cholesterol specifically on their own time. The average doctor has a shockingly low understanding of diet and nutrition in general. They're great for general paintbrush prediction, trauma, acute conditions, etc. But don't do well with people who don't necessarily match the norm. Such as HFLC eaters.

@jw-vx8im

3 жыл бұрын

It's good we can access labs that will test our blood and no need for a Dr visit

@gabriel65304

2 жыл бұрын

@@joeohalloran9309 and what you did? Thanks 🙏🏻

Brilliant presentation. Short and precise. I was at first surprised re Prof Sikaris' opinion re statins but he only refers their use to a very small population. Other specialists out there are very cynical and negative regarding use of statins, and women in particular should never ever consider taking them. I have learned quite a lot from this presentation. Thank you!

@santinorider7536

3 жыл бұрын

@Metanoia Highly debatable.... ebm.bmj.com/content/early/2020/07/05/bmjebm-2020-111412

@anachav3072

3 жыл бұрын

Statin was authorized when the great well intended people from Merck lab - requested authorization for people for hipercolesteronemia, after the trials were stopped because they showed statin was causing tumors in the lab .. please read how this drug was developed.. scary!!!!

@maureengreen8240

11 ай бұрын

Why should women not consider taking Statins please?

@SallySturman

6 ай бұрын

Why shouldn't women in particular never take them?

Wonderful and insightful lecture Dr. Sikaris. I wonder what the 'scientist' who voted this down based his or her objections on?

Seems to me insulin resistance and oxidative damage is causing CVD.

@tenminutetokyo2643

3 жыл бұрын

Insulin drives fat storage and too much insulin drives too much fat storage. CVD is massive fat overstorage due to massive high insulin which is triggered by high carb, high sugar diets - which is exactly the diet the AHA recommends.

Thank you ! For sharing your info ! It helps us everyday people to take our health in our own hands & help or self 🙏

Respected Sir one of best on cholesterol CRUX 👍

I come from Singapore which is supposed to have high standards of medical practise. But people like myself with moderately raised LDL (3.3mm/L) and normal HDL (1.0 mmol/L) moderately raised TG (2.1 mmol/L) are prescribed statins. None of the doctors told me about LCHF.

@carrollhoagland1053

7 жыл бұрын

Hong Kong and France have the lowest rates of heart disease in the world ... they eat meat and cheeses and not much sugar, ... in this day and age you must take charge of your own health .. whereas chips and dip ... high carbs ... 70 Going On 100 ... Keys falsified his data, very simple ... will go down as the "Worst Scientist in History" ...

@kiril1

6 жыл бұрын

Don't you know yourself, that a low saturated diet should at least a little bit reduce your LDL? Why not trying this? But maybe yur doctor knows better, maybe you have a history of heart disease in family, at pretty young age, and he's decided it's better for you to start statins. As for an average "healthy" man, LDL 3.3 is a borderline, with an almost normal HDL, but a high TG, so maybe a diet would help you. But your doc knows better, I believe,

@boabdilyad5441

3 жыл бұрын

I’m from singapore too and I stopped statins since 2013 and doing intermittent fasting since 2013 and Low carbs and at times whole foods plant based . Low triglycerides and the rest high and I feel so good and my blood sugars are back to normal range after years of struggling . Statins was the worse gave me frozen shoulders , muscles aches and feeling of laziness the moment I stopped a week later I begin to feel better . My poly now once a year is just for my subsidies blood test and it’s all good reading , thankful for Low Carbs and Intermittent Fasting lifestyle

Excellent!

We need more talk of LMHR fenotypes. I am very interested in this area particularly. My TG/HDL ratio varies. Now it is 0.34 but has been lower too. There are quite many of us who like to manipulate these numbers by changing diets and fasting. I've done quite a bit of experimenting to drive the local blood labs mad (not to be rude, but for biohacking self interest ;)

Thank you Dr Sikaris, I've been avoiding to take statin ( since a badddddd experience with it) I learned from a Ana Ma La Justicia - degree in Chemical Science- that Lecithin+ vit C had clear plaque on people she advised. Is my hope for research on it come up soon , would you take on it?

The cholesterol rises to combat inflammation in the body. Lowering inflammation in the body I think would help balance the cholesterol levels. Like omega 3/6 etc. Just my thoughts. Have a great day!

@jameskantor0459

4 жыл бұрын

Robdrums Studios , do you take fish oil, or what have you been do to lower your inflammation.

@ellenorbjornsdottir1166

4 жыл бұрын

Combat, no, it's to create "euflammation" (inflammation that's good for you). Antiinflammatory drugs and certain lectins create misflammation.

@pilotrtc

2 жыл бұрын

@@ellenorbjornsdottir1166 That is very interesting. I didn't know there was good inflammation. Is there a reference or study you can share that explains this more? Thank you in advance.

I've always had high cholesterol . I'm in keto for about six weeks and LDL and total amount has gone right up . My HDL has gone down and tryglicerades gone up which I didn't expect . When I did keto in February everything went down but is only been on keto a few weeks . Should I give up keto . Mry total is 373. 9.69 the highest ever . LDL 7.34.

You’re a special man 👨

Dear Dr Sikaris , Thanks for a great presentation [ when to worry ] , you are helping many of us that have been misinformed for too long . I have a question however . I have been on LCHF for about 9 months , lost 20kgs but I think I may be a hyperesponder. Total chol 9.7 , trigs 1.3 , HDL 1.2 , LDL 7.9 , non HDL 8.5 Chol/HDL 8.1 , Trig/ HDL 1.08 , hbA1c 5.1. In this presentation you say that we need to learn more about Lp[a] and PCSK9 .My question is , since making this video 3 1/2 years ago have you formed an opinion about these . From the above numbers should I be worried , do you think I would have modified LDL and what do you think about ceasing statins in my case. As a disclaimer I accept that nothing you say will be taken as medical advice and I release you from any responsibility for any consequences to me. Thank you in advance for taking time from your busy day . Regards Brett

@gabriel65304

2 жыл бұрын

my cholesterol levels after 2 months of keto diet, august 2021 HDL- 50 LDL - 275 Try. - 85 Man 53 old, I got a stent in February 2021 , I have stopped all medication after 1 week. Is dangerous my cholesterol? Thank you for your help

Any developments regarding Lipoprotein (a)? From memory, it appears to fulfill a role in collecting up and creating a malleable / billowy partially stable puff around a site of arterial inflammation, so that blood can still flow about and does not tear off this preliminary 'scaffolding" - thanks to those aforementioned mechanical properties conferred by Lp (a) which permit it to yield to the fluid motion ...

I've got hypothyroidism (Hoshimoto). My Levothyroxine dose was changed from 150ug to 112ug, I was overdosed with Levothyroxine for 5 years. My thyroid levels are now normal and I feel fine but my LDL went HYPER on LCHF, off-the-charts high. Before the dose change my LDL was normal for LCHF. Something to consider, the role that the thyroid plays in LDL.

@nolanerunner1

10 ай бұрын

Thanks for reminding me I remember reading about this but forgot to mention it to my gp who wants me on statins despite heart being fine

@KrummyProductions

9 ай бұрын

Mike, I kicked hypothyroidism by stopping eating gluten, and all grains. Check out Dr. Osborne here on KZread.

Should bread and all forms of sugar be cut out of our diet?

I'm no doctor but after watching this lecture I feel I know a lot more about the subject than my local GP. Not really but I feel there's a massive gap in his knowledge of the subject.

Is there a follow up lecture to this yet?

Wow! Very informative.. I live in Greece and my husband who has always eaten "healthy" (low fat,never fast food,etc) Hdl:56 Ldl:280 normal triglycerides,,, needed a triple bypass and operation on carotid.Should mention he WAS a smoker. Recovered extremely fast . He is now on statins and following a low fat diet. I believe he eats too many carbs. He rarely indulges in sweets but bread with every meal and pasta once a week. The famous Greek white bean soup and lentil soup once a week. Etc...healthy food. I am a low carber...but until he hears the order from the doctor he will continue eating carbs. I cannot find any new information regarding low carb high fat after heart surgery. Everything is preventative information....Would it be possible to shed a little light on this subject.? thank you...and thank you for posting this video....

@kiril1

6 жыл бұрын

With that high LDL and some amount of nicotine(smoking), and maybe some family history of heart disease, its' no wonder he's a heart disease(CABG) himself.

A wonderful video. I have some question though. Does high uric acid cause LDL to become oxidized?

@robertwillis1819

3 жыл бұрын

By definition high uric acid levels would indicate a liver that is having to process high levels of fructose and or alcohol. Can't prove directly that it would cause oxidation of ldl but high uric acid levels would encourage insulin resistance, inhibit nitric oxide and raise blood pressure which on a standard diet would lead to higher glucose levels in the blood and increase chances of oxidised ldl.

Thumbs up and subscribed!

I’ve been following this topic for a couple of years now, it is clear that there is considerable disagreement among experts as to whether LDL particle size is a significant factor in atherogenesis. For example, Peter Attia had Tom Dayspring on his podcast, and Tom says that all LDL, IDL, and even most VLDL can get through the endothelial wall. In other words, having mostly large, fluffy LDL won’t save you.

How do you know if you have FH? Is there any other marker than just a high LDL-C?

Would be useful if the cholesterol and LDL and Triglyceride values were also given in units used in the USA.

@paulinemcgee351

6 жыл бұрын

Just go to CholesterolCode.com and punch in your numbers and it will convert it for you

@cinnamongirl3070

4 жыл бұрын

conversion chart- www.heartuk.org.uk/downloads/health-professionals/factsheets/cholesterol---triglyceride-levels-conversion.pdf

Thanks Dr. Sikaris ... as lipid diagrams are good and help ... also a big fan of Ivor Cummings ... as like the Engineering approach to solving problems since that is what it is all about, something that Medical doctors have forgotten ,,, due to bad training ... I am zeroing in on the TR/HDL formula as best indicator ... 70 Going On 100 ...

So from what I have found online regarding conversion of triglyceride levels, is it true that 430 mg/dL is very high??

@jameskantor0459

4 жыл бұрын

maria G , yes 430 mg / dL. Do try a lchf diet. It brought my Triglycerides down to 66 mg/dL in 6 months. I was about 170 mg/dL

@techknowledgestuff

2 жыл бұрын

Is this on fasting or non-fasted state? On fasting this is high. On non-fasting, this may be around upper bound of the range. Assuming 430 on fasting, please take appropriate action by correcting diet. Eat fewer meals. Cut the carbs. Stop eating processed/junk food completely.

How do these numbers look in the United States? Thank you, I’ve been keto/carnivore for about4 months and my total cholesterol has gone up and making my doctor have fits about it and wants me to take statins which I am not with do at this point! How can I know if I’m in danger?

What causes small dence LDL? Why not cure the cause of low fence LDL. Why lower by statin drugs if glycation is caused by sugar.

What is the root cause of plaque? Isn't LDL preventing soft liquified plaque from bursting?

Thanks for this. Can someone please direct me to the studies showing that lower cholesterol correlates to higher rates of morbidity and that high cholesterol does the opposite? Thanks!

@reinerschafer1708

3 жыл бұрын

pubmed.ncbi.nlm.nih.gov/33293274/ Nadir of all cause mortality seems to be about 3.6 mmol/L

@gabriel65304

2 жыл бұрын

@@reinerschafer1708 my cholesterol levels after 2 months of keto diet, august 2021 HDL- 50 LDL - 275 Try. - 85 Man 53 old, I got a stent in February 2021 , I have stopped all medication after 1 week. Is dangerous? Thank you for your help 🙏🏻

my LDL is 41 and HDL is 70, total cholesterol is 129. Is this ok. I do not have any other problems.

@gabriel65304

2 жыл бұрын

Perfect

He didn't explain how we measure our sdLDL level (or I missed it.)

At 13:21 you state "if your triglyceride level is below one...they don't have any small dense ldl". I have had two NMR Lipo Protein Tests and my triglycerides measured at .89191mmol/l or 79 mg/dl. and my small LDL-P is 543 nmol/L. I have been LCHF for a year now. What would explain my small LDL-P count? My LDL-P is 2368 nmol/L. Cheers!

@joeohalloran9309

2 жыл бұрын

I have the same question and problem. My triglycerides was 0.9. Yet i have sdLDL My triglycerides/ HDL ratio was 0.7 well below 1.5

Confirms that I should stay on my lchf diet.

@jerseyjim9092

3 жыл бұрын

Did you have a LDL subtraction test?

@gabriel65304

2 жыл бұрын

I don’t know, but sure statins kills you faster then any diet

In US measurement: a triglyceride of 430 mg/dL equals WHAT compared to the 1.5 + triglyceride reading he was talking about as dangerous? I’m Confused about the measurements.

@timsmith3689

4 жыл бұрын

430 mg/dl = 4.85 mmol/L www.omnicalculator.com/health/cholesterol-units

According to Robert Lustig "Metabolical" statins do not work on small dense LDL, and the risk of a heart event is only 1% lower.

I did nt get it. I ate more cheese and butter to increased my Fat intake. And i ate like this for a month and then took a blood test. I took a blood test 8hr later after dinner. Took it in the morning. And my results came . My LDL increased just a bit , My HDL was low and my triglycerides increased two folds. I knew that i am in trouble. I was"nt eating grass fed butter here. I need help . What am i doing here ?

@dr.proteomix1257

Жыл бұрын

Triglycerides should go down significantly when eating low carb. If that doesn’t help try intermittent fasting or even longer fasting if need be. Also consult your doctor, especially when fasting. It is very important to keep your electrolytes in check.

Hi Dr Sikaris, Very interesting presentation, thank you. I am a health science student aiming to increase my understanding of this topic. I thought I'd post you a question in case you read the responses here. :) I did a short course with Associate Professor Ryan Bradley ND, a cardiovascular specialist who has done a wide variety of clinical and observational research including cardiovascular epidemiology in the Multi-Ethnic Study of Atherosclerosis (MESA) cohort. My question to you relates to your point (at about 8 minutes in) about how the big boyant LDL may actually reduce the risk of heart disease, while sdLDL (solid, dense LDL) increases heart disease risk threefold. So bascially, you're saying its not LDL that causes heart disease but purely sdLDL. I am wondering what you think of the conclusions of this study (which contradicts that point)? "LDL particle subclasses, LDL particle size, and carotid atherosclerosis in the Multi-Ethnic Study of Atherosclerosis (MESA)". www.atherosclerosis-journal.com/article/S0021-9150(06)00259-0/abstract The linked research shows that BOTH small and large LDL are significantly associated with subclinical atherosclerosis and carotid intima-media thickness (IMT) independent of each other, of traditional lipids, and of other established risk factors. It mentions that studies showing that small LDL is mainly associated with greater atherosclerotic risk, do not adequately quantify the number of small versus large particles. Particle quantity important to quantify, as explained in this study: "LDL PARTICLE SIZE: DOES IT MATTER?": www.athero.org/commentaries/comm564.pdf : Quoting that study: "small LDL particles contain substantially less cholesterol than large ones, such that at the same serum concentration of LDL cholesterol (LDL-c), individuals with predominantly small LDL have greater total concentration of LDL particles than those with predominantly large LDL". So what is saying is that there is a significant inverse correlation between small and large LDL particles. It apparently has been demonstrated that the relationship of LDL size with cardiovascular disease is confounded by this inverse correlation of LDL particle numbers, and that actually, once this correlation is accounted for, LDL particle size contributes little to increased risk of CVD. Another issue mentioned that further confounds the inverse particle size correlation mentioned previously, is that many studies use 'gradient gel electrophoresis' to measure LDL. This method determines only the distribution of LDL subclasses or average LDL size (large or small) but it does not quantify the number of small versus large particles. Quantifying LDL-p (number of particles) directly can be now be done using nuclear magnetic resonance (NMR) or using apolipoprotein B (Apo B) - also an estimate of the LDL-P. As mentioned in this study, neither of these methods relies on the variable cholesterol content in LDL particles: www.mdedge.com/sites/default/files/pdfs/jfp_labcorpsuppl_final.6.15.16_0.pdf So I guess I am wondering, other than the fact that smaller denser particles lead to atherosclerosis more rapidly, what the point is on focusing on sdLDL in light of the above research showing that in the long term its not just sdLDL that is atherogenic? If high triglycerides are a good proxy for sdLDL, are they not also a good proxy for LDL fullstop? And if so, is the benefit from decreasing triglycerides also because doing so lowers LDL in general? I understand that oxidation comes into play, and antioxidants are useful, but overall, once LDL is deposited isn't it true that its better to just focus on reducing LDL altogether? Apparently this approach can not only stablise LDL but actually reverse atherosclerosis and coronary artery disease altogether... To conclude, this is a (rough) quote of Dr Ryan Bradley's stance on LDL, which is partly what helped spark my investigation into this topic. "My belief based on the science I have that I have reviewed and that has been going on for now for the last 75 years repeatedly demonstrates that the blood concentration of LDL is the leading risk factor for developing cardiovascular disease and having a cardiovascular event. And we know that from many many large studies in cardiovascular epidemiology where we look at cohorts, we know that in animal models, and we know that drugs targeting LDL clearly reduce cardiovascular events and improve mortality. So in my mind it is unscientific to say that LDL doesn't contribute to cardiovascular disease because we literally have decades of research and hundreds of thousands of people that demonstrate that risk". Full youtube of that is here: kzread.info/dash/bejne/iZN8rLZxiaWeqKg.html Thanks, Talia

Wow, I got really interested in your video until you started talking about statins which you need to reconsider. I have familial Hypercholesterolemia and statins just about ruined me. I tried several different ones and Lipitor crippled me up so bad with pain that I could hardly get around. Other statins I tried, my triglycerides got really high. Statins are poisons or are you so blind that you don't realize how many people has had severe side affects and a number of people has died. Anyone who recommend statins instead of alternatives to try, is a fool. And there are other things that cause heart disease as well, including too much calcium and age. In fact there are just as many people if not more people dying of heart disease and their cholesterol levels are normal, even well below normal.

@bigbenhebdomadarius6252

6 жыл бұрын

At the end of the talk, he discusses how the LCHF diet reduces small, dense LDL _without_ drugs, with the implication that statins are therefore unnecessary. Remember that this talk was a small part of a larger conference in which the benefits of the LCHF diet were being discussed. So it was an audience for which the implication did not need to be spelled out. And even for those people who don't see a reduction in sdLDL, there is nevertheless a reduction in triglycerides and a corresponding increase in HDL, which itself lowers the risk of heart disease.

@spfldo

6 жыл бұрын

Agree, statins are dangerous, and were killing me as well. I had terrible side affects, while taking Lipitor and Vytorin e.g., joint pain and excessive muscle cramps, overwhelming fatigue, peripheral neuropathy, and cognitive issues. Most have gone away since discontinuing the meds, but some are permanent, but hopefully not progressive. Time will tell. Carbohydrate/sugar and the TG/HDL ratio is, as the professor says are the most important factors. My TG/HDL ratio used to be 7 to 1 on a low fat diet. On a LCHF (very low carb) diet my HDL more than doubles and my TG dropped by 2/3 and now, I believe my arteries are healing. Here's my story: kzread.info/dash/bejne/naplrayzl67gZbg.html

@MortezaNurcheshmeh

5 жыл бұрын

He mentioned Statin would be good for people with genetic problems where higher amount of sd-LDL is made by body or accumulated in blood.

@CivilianSheepdog631

5 жыл бұрын

100PercentOS2 same here my average total cholesterol is 408 ldl 332 hdl 53 triglycerides 90 and stains of all kinds is nothing but trouble in my body

@mannyradzky493

5 жыл бұрын

100PercentOS2 I took statins for just one year, felt like crap. Stopped 3 years ago, went high fat, low carb. Never felt better.

Dr Sikaris, I think I have familial hypercholesterolemia. My total is 320mg/dl and my LDL is 242, trigs 61 and hdl 66 mg/dl. I'm 31 years old and I've been on the LCHF diet for about 8 months. I suspect modified LDL because my trigs are over 20mg/dl or 1.1 mmol/l. Do I need to start a statin right now!??! I just told me physcision no........... Please respond and thanks!

@calmingdragon2862

7 жыл бұрын

Danny Alford , I realize your comment is over a year old. Can you please tell me what you ended up doing? Taking a statin or not. Thank you very much.

@kristivice5150

5 жыл бұрын

I am also responding really late, but your remnant cholesterol is very low. This is good. This lipid marker correlates strongly with cardiovascular disease. In addition, your trig to hdl ratio is good.

@sofiabriones5708

4 жыл бұрын

This is late reply. Im on keto. Before that my bp was high even I was on vegan, and cant sleep good. My blood works were normal- cholesterol 170, But when Im on keto my cholesterol is 340, ldl 210, triglyceride 67, hdl 70, The good thing is my bp is in normal rate, I sleep good like 8-9hrs. I rather have high cholesterol than not having a good sleep and high bp. Im 62yr old, my hair still black, seldom gray hair which are hiding somewhere lol

Repatha sorts out psk receptors in the blood but costs a fortune

Very informative. You can't help but wonder if it's just the BIG DOLLAR for the Pharmaceutical co.'s that make you worry about this??? Never worried about it when my grand-parents were alive and they lived til a ripe old age!!!!!

@gibbogle

2 жыл бұрын

Their diet had much less sugar and other carbs. They probably ate less overall.

Why aren’t we just testing for small dense LDL?

Great information. But he should have made himself more clear when he was recommending statins. I realize he didn't mean as a form for everyone to lower their cholesterol, but just the mention of that poison needs to be clarified.

@rangerpretzel

4 жыл бұрын

It's not poison for certain folks with certain genetic abnormalities. It's a life saving drug for them. He spells it out very clearly in the video. How much more clear does he have to make it?

The claim that cholesterol in the diet is harmless is clearly refuted by the evidence. So is the claim that karge LDL is harmless. Hiow can he not know this?

Statins are generally not a good Choise DR.

Reduce small dense ldl=lchf no sugar no seed oils

@joeohalloran9309

2 жыл бұрын

No. Not correct. I do all of that and plus fasting omad. I have sdLDL

Cholesterol equals life! But it has two sides, the sunny and the dark. Once it gets to the dark side we have problems!

Sorry no. I've heard presentations by numerous medical professionals that refute this guy's claims that statins reduce small dense LDL count. In fact all statins do is cause more receptors on the liver for healthy LDL particles, which actually increases the concentration of small dense LDL in the bloodstream. Statins make the situation worse, not better. And even for those people who take them, studies have shown that taking a statin for multiple years only increases lifespan by a few days on average. Literally less than a week. And that's with cherry-picked data. I'm surprised and a little shocked that this presenter also never mentioned that small dense LDL is actually caused by oxidation of the healthy LDL particles by blood glucose. Hearing a professional promote statins while ignoring high blood sugar as the primary driver of LDL damage is really quite a head-scratcher to me. Perhaps because this video is 7 years old, he wasn't aware of these facts. But bad information like this only confuses people and muddies what is already a very complex topic.

@Definitely_Someone

Жыл бұрын

Maybe he was probably only talking about the Homozygous FH. Can you please provide me the presentations and data you mentioned specifically regarding that statins dont reduce small dense LDL? im really curious about it

Follow the money thats why! who won't get rich if your healthy?

@janebreaux3530

7 жыл бұрын

sue leedy that's right! Unfortunately one of them is the very doctor that's suppose to be helping you.

@toni4729

3 жыл бұрын

Most doctors don't understand the conection between cholesterol and diet. They don't know that a healthy diet is the cause, quite often of high cholesterol so, they try to control it. It's so sad.

I'm very confused, 🤔 i have just got my blood work back . I'm 62 years old. My triglycerides/ HDL ratio was 0.7. My triglycerides was 0.9 My HDL 1.3 Yet i have sdLDL LD LDL 2: 1.03; & 3: 0.67; & 4: 0.31; My VLDL was 0.7 My LDL was 4.3 My total Cholesterol was 6.0 Mean particle size 260L Why is this happening? I Fast everyday omad and eat low carb foods? Im Devastated!!!!!

@nickbardan3867

2 жыл бұрын

Why?

I can barely hear you speaking.

If you eat meat and butter you remone more sd LDL.

PORRIDGE EVERY DAY NO NEED FOR STATINS

@michaell3711

2 жыл бұрын

Is your porridge sweetened and with what ?

@gibbogle

2 жыл бұрын

@@michaell3711 The Scots used to eat it with salt, no milk or sugar. Not now, though.

@joeohalloran9309

2 жыл бұрын

I'm going to try that, as I've been doing keto, low carb, omad fasting. And it doesn't work for me. Love my porridge and I miss it.

Lost me at statins

@cinnamongirl3070

4 жыл бұрын

Right?!! This video was super interesting, except for his recommendation of statins!

@rangerpretzel

4 жыл бұрын

Too bad your brain shut off when your bias kicked in... He spelled it out VERY clearly that Statins (oh, your brain just shut off again...) Anyway, he spelled out *incredibly* clearly that certain folks have certain genetic abnormalities and *benefit greatly* from these types of LDL lowering pharma drugs by almost completely eliminating their small dense LDL (sdLDL) Without these drugs, these folks have a much higher risk of heart attack and arterial disease.